Interaction between 4-hydroxyphenylpyruvate dioxygenase–inhibiting and reactive oxygen species–generating herbicides for the control of annual weed species in corn

Abstract The complementary modes of action 4-hydroxyphenylpyruvate dioxygenase (HPPD) and photosystem II (PSII) inhibitors have been credited for the synergistic weed control improvement several species. Recent research discovered that reactive oxygen species (ROS) generation subsequent lipid peroxidation is cause cell death by glutamine synthetase inhibitor glufosinate. Therefore, a basis synergy exists between glufosinate HPPD inhibitors, but interaction has not well reported. Four field experiments were conducted in Ontario, Canada, 2020 2021 to determine HPPD-inhibiting (mesotrione tolpyralate) ROS-generating (atrazine, bromoxynil, bentazon, glufosinate) herbicides on annual corn ( Zea mays L.). ROS generators with provided ≥95% velvetleaf Abutilon theophrasti Medik.), except tolpyralate + glufosinate, which was additive at 8 wk after application (WAA) 87% control. Tank mixes plus common ragweed Ambrosia artemisiifolia L.), antagonistic WAA. Tolpyralate barnyardgrass [ Echinochloa crus-galli (L.) P. Beauv.] Setaria spp. Common lambsquarters Chenopodium album L.) WAA mesotrione atrazine, or bromoxynil bentazon. Herbicide tank generally wild mustard Sinapis arvensis WAA, atrazine bromoxynil; however, each mix 97% 100% S. . Results from this study demonstrate co-application controlled all broadleaf >90% exceptions A. C. Mesotrione PSII E. 48 68 percentage points less than respective WAA; grass similarly.